Banana PI M2 Zero — это одноплатный компьютер на базе SoC Allwinner H2+ в составе которого входят: 4 ядра ARM Cortex-A7 с тактовой частотой 1.0 ГГц, двухъядерный GPU Mali400MP2 GPU частотой 600 МГц с поддержкой OpenGL ES 2.0. Мини-компьютер несёт на борту 512 МБ оперативной памяти DDR3 SDRAM.
Banana PI M2 Zero — это аналог Raspberry Pi Zero W от «Lemaker». Снаружи очень похожи, а именно расположением компонентов на плате: 40-контактный разъем расширенного ввода-вывода (GPIO), слот для microSD, интерфейс CSI (Camera Serial Interface) для подключения камеры по интерфейсу MIPI, два разъема micro-USB (один для питания, второй для подключения периферийных USB устройств), разъем mini-HDMI. Также поддерживает Wi-Fi сети стандарта 802.11n и Bluetooth 4.0 (Bluetooth Classic и LE).
В отличии от Raspberry Pi Zero W, у Banana PI M2 Zero более мощный процессор (4 ядра против одного), можно подключить внешнюю антенну, у Raspberry Pi Zero W она является частью макетной платы. Также имеет две кнопки для питания и сброса, светодиодный индикатор.
Питание Banana PI M2 Zero осуществляется от 5-вольтового адаптера через разъём micro-USB или пины питания. Рекомендуется использовать источник питания с силой тока 2 А.
Banana PI M2 Zero поддерживает ОС, основанных на Linux ядре: Raspbian, Ubuntu, Android, Debian и др..
Основные характеристики
- SoC Allwinner H2+;
- Процессор 32-битный 4-ядерный ARM Cortex-A7 с тактовой частотой 1.0 ГГц;
- Графический 2-ядерный адаптер Mali400MP2;
- Память на 512 МБ LPDDR3 SDRAM.
- WIFI 802.11n + Bluetooth 4.0 Low Energy (BLE).
Доступные операционные системы
ОС: Ubuntu 16.04 mate
Скачать файл: 2017-07-21-ubuntu-16.04-mate-desktop-demo-bpi-m2z-sd.img.zip
Технические характеристики Banana PI M2 Zero
| Система на кристалле (SoC) |
Allwinner H2+ (CPU + GPU) |
| Процессор |
32-битный 4-ядерный ARM Cortex-A7 с тактовой частотой 1.0-1.2 ГГц, 32KiB Instruction + 32KiB Data L1 cache на ядро и 512KB L2 cache |
| Графический процессор | двухъядерный графический адаптер Mali400MP2 GPU @ 600 мГц с поддержкой OpenGL ES 2.0, Кэш 2-го уровня: 256 КБ |
| ОЗУ | 512 МБ SDRAM LPDDR3 совместно с GPU (Samsung K4B4G1646D-BCK0) |
| Хранилище | слот (T_CARD) для карты памяти (до 64 ГБ) microSDHC |
| Wi-Fi/Bluetooth | Wi-Fi 802.11n и Bluetooth 4.0 (Bluetooth Classic и LE), обеспечиваемые микросхемой AP6212 (возможные варианты AP6181, AP6335); порт для подключения антенны (WIEI) |
| Видео вход | 1 порт Camera Serial Interface (CSI): Поддерживает 8-битные YUV422 CMOS Датчик Интерфейс Поддерживает CCIR656 протокола для NTSC и PAL Поддержка SM пикселей камера Датчик Поддерживает видео захвата решение до 1080P @ 30fps |
| Видео выход | HDMI через разъём mini HDMI (1080p60) |
| Аудио вход | Через I²S |
| Аудио выход | mini HDM |
| USB-порты | 1 порт micro-USB 2.0 (OTG) |
| Периферия | 40 портов ввода-вывода общего назначения (GPIO), UART, I²C/TWI, SPI; пины питания: 3,3 В, 5 В и земля; 3 портов Debug UART и земля (GND RX TX). |
| Питание | 5 В, 2 А через порт micro-USB (PC_IN) или GPIO |
| Кнопки | питания и сброса |
| Индикаторы (LED) | питания |
| Размеры | 60.0 мм x 30.0 мм |
| Вес | 15 г |
| ОС | Raspbian, Ubuntu, Android, Debian |
Распиновка — GPIO (General purpose input-output)
Banana Pi BPI-M2 Zero demo: 1080P video play , glmark2-es2 , wiringpi
Добрый день.
Есть banana pi m2 zero и есть ИК датчик TSOP2136.
Не подскажете как прочитать данные от этого датчика, используя wiringpi?
Читая данные с пинов wiringpi, мы имеем всегда 0 или 1, а с этого дачика нужно читать зачение кнопки нажатой на пульте (это больше двух значений).
Если ещё актуально) используйте ацп
This intake was considered as a part of their normal diet and was accounted for
in analysis of dietary intake. In a post-stroke muscle loss mouse model, PINTA-745 demonstrated a big enhance in muscle mass, energy, and motor function [68].
MSTN’s relationship with muscle growth has led to the widespread research of its inhibition for the remedy of muscle, bone, and metabolic illnesses, as nicely
as enhancing agricultural meat production [8–11]. Medical trials using MSTN inhibitors started in the early
2000s, primarily aiming to increase muscular function and survivability
in muscular dystrophies. Despite achieving widespread success in preliminary animal trials,
the journey to market for MSTN-based medication has been largely disappointing, as not one of
the clinically examined inhibitors have been permitted for mediating
muscle mass [12–14]. Though the scientific failures of
muscular dystrophy-targeting medication have branded MSTN inhibitors
as a fruitless endeavor, recent advancements in inhibitor software and design supply promising prospects for creating viable MSTN
therapeutics.
This ingredient helps in growing the levels of follistatin and keeping myostatin levels low.
If you want to know the means to lower myostatin blocker, just bear in mind that there’s an essential compound within the
muscle-building course of known as follistatin. It regulates myostatin-signaling pathways, leading to
lower motion of myostatin inhibition that results in extra muscle
fiber constructing.
Which antidepressant is greatest for you depends on a
number of elements, corresponding to your symptoms and another well being
situations you might have. All SNRIs work in much
the identical way and generally could cause the identical sorts
of side effects. Aspect effects that do occur are often gentle
and go away after the primary few weeks of remedy. If you possibly can’t handle the unwanted effects of
1 SNRI, you may have fewer unwanted side effects with a special one, as every SNRI has a special chemical makeup.
Leptin signaling, by way of the hypothalamic melanocortin receptors, acts as a attainable mediator of inflammation-induced cachexia and anorexia in CKD.
Kidneys clear the circulating leptin, therefore, its
serum focus is elevated in uremia and CKD [143].
Leptin is taken into the brain by the blood-brain barrier (BBB) and binds to the
leptin receptor, suppressing meals intake by inhibiting the arcuate nucleus
of the hypothalamus. Leptin controls cachexia by
way of modifying energy expenditure and food intake by way of the hypothalamic melanocortin system [21].
In CKD patients, the decreased renal function results in an elevation in leptin levels,
probably as a outcome of diminished renal
clearance, thus, elevated levels of leptin are related to poor nutritional statuses, similar to hypercatabolism and low serum
albumin [9, 144]. In the hypothalamic arcuate nucleus neurons, Proopiomelanocortin (POMC,
a pro-peptide precursor) is produced that impedes power storage and meals consumption by releasing α–melanocyte-stimulating hormone (α-MSH) [145].
Α-MSH prohibits food intake by binding to melanocortin receptors-3 and − 4 (MC3-R and MC4-R), primarily by way of
MC4-R [134].
Furthermore, Smad2/3 phosphorylation by myostatin triggers atrophy in adult muscle fibers partially through upregulation of an ubiquitin ligase
atrogin1 [36]. In addition, through a Smad2-dependent elevation of nuclear factor of
activated T cells 1, myostatin was recently described
to instantly speed up the formation of osteoclasts from bone marrow-derived macrophages, rising
bone resorption [7]. For one, epicatechin seems to extend aerobic and
anaerobic health by bolstering mitochondrial biogenesis and nitric
oxide manufacturing [8].
Sarcoplasmic hypertrophy is when glycogen storage will increase the size of the muscle cell, making it primarily short-term progress, meaning if the myostatin level were to return to regular, the glycogen in the muscle would lower and mass can be misplaced.
Venous blood from the antecubital vein was collected
into a serum separator tube utilizing a Vacutainer apparatus and needle
(Becton, Dickinson and Company, Franklin lakes,
NJ). Immediately after blood draw, blood samples remained at room
temperature for 20 min to clot, and then had been centrifuged (1,000 g) for 20 min to separate serum.
Serum samples have been aliquoted into 1.5 mL tubes and instantly frozen at −80°C for
the later analysis. Percutaneous muscle biopsies (20–25 mg) have been obtained from the center portion of the vastus lateralis muscle at the midpoint between the patella and the larger trochanter of the femur at a
depth between 1 and 2 cm based mostly on previously-used procedures (20).
The identical leg and common location (determined by pre-biopsy markings) was biopsied at each testing session.
Topics signed an informed consent that was permitted by the Institutional Evaluation Board for Human topics previous to participation. Each topic completed
a private info sheet and a regular medical history
form verified by a registered nurse. Subjects had been free from any main metabolic problems (i.e.
heart illness, diabetes, thyroid disease, etc.) in addition to major musculoskeletal problems that may intrude with
their ability to exercise and/or complete the tests through the three
testing periods. Subjects weren’t permitted in the study if that they
had taken any self-reported ergogenic dietary dietary supplements (i.e.
creatine, androstendione, myostatin inhibitors, pro-hormones, and so forth.)
within six months previous to the onset of the examine.
AICAR was additionally reported to enhance exercise capability
in mice by ~44% but has poisonous effects [14].
The development of novel and safe therapies is thus, warranted given the typical older population profile
of topics with sarcopenia and likely indefinite period of treatment.
Epi is a naturally occurring flavanol present in cacao and green tea and has been orally ingested safely by people for
many centuries.
Testing of myostatin inhibitors in animal fashions of muscular dystrophy [34] has
shown generally positive effects on muscle mass
however inconsistent effects on muscle operate and histopathology [reviewed in [35▪]].
ActRIIB-Fc or ActRIIB shRNA given to mdx mice, a properly
used but not perfect mannequin of human muscular dystrophy [36,37▪▪],
produced will increase in muscle mass and total pressure but specific pressure
was unchanged [38,39▪,40]. In distinction, a current examine reported
a rise in specific pressure of the soleus muscle in mdx
mice after long-term exposure to a myostatin propeptide [41].
Studies with myostatin inhibitors have not proven any enchancment
on eccentric contraction-induced pressure drop, a key measure of myofiber structural integrity [40,forty two,43].
Therefore, exploiting phytoestrogens can exert a significant anti-inflammatory effect, which might be useful in cachectic CKD patients.
A high dietary fiber content material and ω-3 fatty acids of fish oil significantly eicosapentaenoic acid can reduce pro-inflammatory cytokines (i.e.
IL-6 and CRP) ranges in non-renal sufferers [7, 16].
However, it was reported that fish consumption in dialysis sufferers could decrease the mortality fee (approximately
50%) in the course of the observation period [167].
In addition, the failure to detect a statistical difference in LOAD mobility
scores between the placebo and therapy teams could replicate
a scarcity of power to recognize a distinction between the 2 teams.
With comparable outcomes, a future study would need 50 canines
in every group to identify a statistical difference at each
time point. It’s made in his world-class NutraBio facility, but
with Unbound and supplements like BYLD,
you’ll get the next era of components. When it involves muscle constructing supplements, that’s a big
deal, as a result of most of us already learn about protein, creatine, betaine,
and similar ergogenics. They say it keeps their strength the same,
their physique composition has not modified, and they’re very lean with 6%
physique fats.
Throughout the examine, four muscle biopsies were collected (days
1, 14, 28, and 42) from the vastus lateralis of the leg
that was assigned for immobilization. The
first biopsy was collected from roughly 15 cm above
the patella, and the following biopsies have been collected
from ~2 or 3 cm apart between them to keep away from the impact of pre-sampling.
All muscle biopsies were obtained using a 5-mm Bergstrom needle, custom
modified for handbook suction beneath 1% xylocaine local anesthesia.
By Way Of handbook dissection, muscle tissues had been rigorously free of visible connective tissue, fats, and blood.
A piece (~40 mg) of the muscle tissue was embedded
in optimum chopping temperature compound (OCT, Tissue-Tek,
The Netherlands) for immunohistochemical evaluation. The remainder of the muscle tissue was snap-frozen in liquid nitrogen for gene and protein expression evaluation.
This results in increased and speedy muscle
progress even beyond the body’s pure capacity to do so. This subsequent blocking of myostatin by follistatin 344 results in the suppression of myostatin exercise.
This subsequent blocking of myostatin by follistatin 344 leads to the suppression of myostatin activity (2).
The myostatin signaling pathway and its function in regulating skeletal muscle has been just lately reviewed [23,24].
The ingredients are thought to reduce back myostatin’s level in the
body to increase the speed at which muscle is constructed.
Evidence Based Mostly Muscle is a trusted source for scientifically-backed fitness recommendation. The platform focuses on varied elements of fitness, including muscle acquire, nutrition, supplements, and methods to get ripped.
Due To This Fact, there may be increasing evidence that myostatin inhibitors
can enhance muscle function within the mdx mouse through a rise in muscle mass and whole pressure but do not persistently improve the underlying weak spot of dystrophic
muscle. It has been suggested that the degree of muscle disease on the time of remedy could affect outcome [43].
Human muscular dystrophy disorders display paradoxical muscle wasting and
selective hypertrophy of skeletal muscular tissues, leading to imbalance, contractures and postural instabilities [37▪▪].
Dysferlin null mice, a mannequin of dysferlin-deficiency
muscular dystrophy [48], expressing the myostatin inhibitor follistatin, demonstrated a transient increase
in muscle mass adopted by decreased muscle mass
and function and increased muscle fibrosis [Lee et al.
Another mice-based medical study suggests that follistatin prevents the method of metastasis in lung and ovarian most cancers. Animal-based trials centered on this glycoprotein recommend that it considerably improves muscle development and supplies a realm of secondary benefits. Kim and colleagues investigated the results of the high-fat, high-energy food regimen (HFED) regarding reasonable protein restriction in a rat mannequin. Their findings demonstrated that rats with massive kidney ablation using a high-fat, high-sucrose food plan might usually gain weight and confirmed a lower in proteinuria and inhibition of oxidative stress in addition to irritation within the remnant kidney [97].
Total RNAs from cells muscle and adipose tissues have been extracted
using Trizol reagent (Thermo Fisher Scientific, Waltham, MA, USA).
In addition, the mRNA and protein of FMOD, MSTN, and adipogenic markers (CD36, PPARγ, and CD163) were considerably decreased in Ac-MIF1-treated cells, and FMOD and PPARγ mRNA and protein expression were
decreased in Ac-MIF2-NH2 (Figure 7B). Furthermore,
Smad2 and Smad3 mRNA expression was decreased in Ac-MIF-treated cells (Supplementary Determine S12).
MSTN protein effects have been observed in C2C12 cells after 2 days of therapy in proliferation or myogenic differentiation media.
Cell proliferation and fusion indices have been decreased
by MSTN protein therapy (Supplementary Determine S1).
Myokines are identified to be concerned in MSC activation and regulate their main capabilities,
for example, they augment proliferation and differentiation charges (Mandai et al., 2017).
Intracellular aggregation of methylglyoxal, a precursor of superior glycation end-products
(AGEs), and subsequent glycation of biomolecules impaired ECM
reworking, and curcumin and gingerol have been reported to
reduce the impact of AGE on myoblasts (Baig et al., 2017).
Furthermore, enhanced AGE manufacturing and consequent RAGE (AGE receptor)-AGE interaction hinders the muscle
development program. We also found by in silico analysis
that the MSTN-ACVRIIB interplay is lowered by curcumin or gingerol.
Protein-ligand (curcumin/gingerol and MSTN) and protein-protein interactions (MSTN and ACVRIIB) studies had been carried out to explore the
impact of curcumin and gingerol in the myogenesis processes.
Nonetheless, the free energy of MSTN to ACVRIIB binding
fell to −46.55 and −47.26, correspondingly, for MSTN-curcumin and MSTN-gingerol complexes, exhibiting
that curcumin and gingerol intervene with MSTN-ACVRIIB
interaction (Baig et al., 2017). Other TGF-β superfamily members,
along with MSTN, are documented to be efficient adverse muscle
regulators, notably «activin A» being the second adverse muscle regulator.
Moreover, the stimulation of Sirtuin 1 (SIRT1) and NRF2 signaling techniques has been documented to mitigate mitochondrial harm and enhance kidney activity, proteinuria, histological alterations,
and irritation in aged mice. Further analysis is required to analyze the attainable impression of resveratrol on protein-energy wasting among people with CKD.
This study provides the first direct proof of metabolically helpful results of Urolithin A in main human skeletal
muscle cells.
Thus, taking an epicatechin supplement can ostensibly enhance skeletal muscle mass, energy, and endurance.
Myostatin is a human progress factor that prevents extreme
muscle progress, and abnormally high ranges can cause the lack of
muscle mass. Regardless Of the shortage of proper data, myostatin has become
a sizzling subject amongst athletes and bodybuilders,
who claim that inhibiting it might possibly enhance muscle
progress. Skeletal muscle is a plastic tissue that
adapts its structure and metabolism in response to a
quantity of inner and exterior variables, significantly mechanical load and protein ingestion [1].
Loss of muscle mass and function happens rapidly with muscle disuse inherent to bed rest and single-leg
immobilization [2, 3], which results in impaired useful capacity,
insulin resistance, and an increased danger for morbidity and
mortality [4].
References:
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